Apostolos P. Georgopoulos, MD PhD
Director, Brain Sciences CenterBrain Sciences Center (BSC)
Regents Professor, University of Minnesota
American Legion Brain Sciences Chair, University of Minnesota
McKnight Presidential Chair in Cognitive Neuroscience, University of Minnesota
Professor of Neuroscience, Neurology, and Psychiatry, University of Minnesota
Elected Member, National Academy of Medicine (2004)
Elected Member, American Academy of Arts and Sciences (2002)
Editoral Board, Vaccines
Researcher, Minneapolis VA Medical CenterVA Medical Center (VAMC)

omega@umn.edu

Education
MD, University of Athens, Athens, Greece
PhD, University of Athens, Athens, Greece

Specialty/Focus
Human Leukocyte AntigenHuman Leukocyte Antigen (HLA)Genes that are located in the Major Histocompatibility Complex (MHC) of chromosome 6 and play a central role in immune recognition. Most investigations of association of HLA to various diseases have focused on evaluating HLA allele frequencies in diseases of interest, as compared to the general, healthy population. Such studies have demonstrated HLA involvement with cancer, autoimmune, and in- fectious diseases. HLA Class I proteins (HLA-A, B, C) are expressed on all nucleated cells and present peptides from endogenous proteins to cytotoxic T lymphocytes engaged in immune surveillance. HLA Class II proteins (HLA-DRB1, DRB3/4/5, DQB1, DPB1) are expressed on antigen-presenting cells and present peptides derived from exogenous proteins to CD4+helper T cells. A previous study of Gulf War syndrome in 27 veterans found that HLA DRB1*15 was more prevalent in cases than controls with an odds ratio of 1.66, although this association was not statistically significant.
Vaccines
Gulf War IllnessGulf War Illness (GWI)Shortly after the Gulf War (1990-91), veterans started to report a variety of health problems that began during, or soon after returning from, deployment, prompting investigation into the epidemiology and etiology of the complaints. Those investigations revealed that diffuse symptoms such as fatigue, musculoskeletal pain, mood and neurocognitive complaints, gastrointestinal problems, and rashes were most commonly reported. The constellation of symptoms, now commonly referred to as Gulf War Illness (GWI), has affected a substantial number of Gulf War veterans. Several population-based studies have demonstrated that these symptoms occur at significantly higher rates in deployed Gulf War veterans relative to their nondeployed peers and other veterans, raising the issue about possible in-theater exposures and stress as contributing factors. However, these symptoms are also present in non-deployed military personnel, leading some to suspect other causes, including reactions to vaccine adjuvants. In summary, GWI is now a recognized constellation of symptoms of unclear etiology, also co-occurring with psychiatric disorders.
Dementia
Healthy Brain Aging
Magnetoencephalography
Neural mechanisms of cognitive processes
Neurophysiology of motor control and cognition
Functional MRI of motor and cognitive processes

Publications

Pages: 1August 2024 through July 20222July 2022 through April 20213February 2021 through November 20194October 2019 through January 20175November 2016 through July 20146January 2014 through April 20107January 2010 through December 20068December 2006 through July 20049July 2004 through January 200210October 2001 through March 199911March 1999 through November 199612July 1996 through May 199413March 1994 through December 199114December 1991 through August 198815August 1988 through August 198316August 1983 through July 1975
  1. The number of cysteine residues per mole in Apolipoprotein EApolipoprotein E (ApoE)a plasma lipoprotein discovered in 1973 (Shore and Shore 1973). It binds low-density lipoprotein receptors, thereby facilitating cellular lipoprotein exchange and metabolism. The human apoE polypeptide consists of 299 amino acids and comprises three polymorphisms resulting from single amino acid substitutions. Three isoforms (E4, E3, and E2) are the result of cysteine^aEUR"arginine interchanges at two sites, namely residues 112 and 158; however, other genetic variants have been described. These three isoforms, each differentially affecting protein function, result in six phenotypes: three homozygotes (E4/4, E3/3, E2/2) and three heterozygotes (E4/3, E4/2, E3/2). With respect to the number of cysteine residues per mole, E2/2 contains 4, E3/2 contains 3, E4/2 and E3/3 each contain 2, E4/3 contains 1, and E4/4 contains 0. The number of cysteine residues per mole (CysR/mole) provides a numerical, biochemical scale in lieu of the genotype-based categories. affects systematically Synchronous Neural InteractionsSynchronous Neural Interactions (SNI)Zero-lag partial correlations in pairs of MEG time series and denote the strength and polarity (positive or negative) of neuronal interactions. Anomalies in SNIs as assessed by MEG differentiate psychiatric disorders from healthy brain functioning and can discriminate among various brain diseases. From this research, a highly distinctive, unique PTSD SNI signature characterized by miscommunication of temporal and parietal and/or parieto-occipital right hemispheric areas with other brain areas has emerged. These findings, in addition to the growing research applying MEG to other psychiatric disorders, highlight the utility of MEG in identifying biomarkers of disease and underscore the potential for broader clinical applications of MEG. in women's healthy brains Experimental Brain Research (2013, May) Leuthold A, Mahan M, Stanwyck JJ, Georgopoulos A, & Georgopoulos AP